What is T3 Method

Why Single Methods Fail ?

Most of the available methods for treatment of ED are aimed at correcting only one of the causes of ED. As we know now, Erectile Dysfunction is always multifactorial in nature , treating a single cause never provides complete and long lasting solution. Further single mode therapy does not make an attempt to correcting underlying cause but only tries to provide short term solution .Most of the available therapies only target biochemical imbalance and never make an attempt to correct vascular and neural factors. Reliable solution to ED can be provided only if attempt is made to correct vascular ,neural and biochemical factors. This is where our properietory ‘ T3 Method ‘ scores as this multimodal therapy targets all three at same time .


Before initiating any treatment , we carry out detailed evaluation.
T3 Method has three components namely


WAVE makes use of focused energy which interact with the targeted deep tissues where they cause mechanical stress and microtrauma. This stress and microtrauma (also known as shear stress) induces a cascade of biological reactions that result in the release of angiogenic factors which in turn triggers neovascularization(angiogenesis) of the tissue with subsequent improvement of the blood supply WAVE is a revolutionary treatment of ED, and probably possesses unprecedented qualities that can rehabilitate erectile tissue.

The clinical improvement in subjective erectile function together with the significant improvement in penile hemodynamics following WAVE confirm that it has unique properties .It is both non invasive and tolerable and without any adverse or unwanted effects. Its main advantage is its ability to improve and potentially restore erectile function in men with ED without additional pharmacotherapy. Hence, WAVE is an appealing addition to the armamentarium of existing treatment options for ED.

Reliable solution to ED treatment
methods for treatment of ED
treatment method of male ED

This concept represents a new modality for the treatment of ED: a neurophysiologic mechanism for the stimulation of erection, not just an effect at the vascular response level. Thus, VANE  adds to current ED technologies, offering an alternative approach that more closely exploits and mimics normal biologic erections.

From a neurophysiologic viewpoint, penile erection is a culmination of multiple successful nerve reflexes which then initiate a vascular event. Erection is controlled by spinal autonomic centers, the activity of which is dependent on input from supraspinal centers and the genitalia. Sensory information, captured by receptors in the penile glans and shaft, is relayed via the dorsal nerve of the penis and perineal nerve to the afferent pudendal nerve, which is then relayed to the spinal cord. Efferent stimuli are subsequently transmitted to the penis via the cavernous nerves. Of additional anatomic importance, it is known that the pudendal nerves directly connect with the cavernous nerves at the base of the penis in 70% of cases. Genital afferent nerve vibratory stimulation can lead to re exogenic gradual filling of the penis with arterial blood by activating the pudendo- cavernosal re contraction of the perineal muscles via activation of the bulbocavernosus reflex (BCR), which contributes toward erection rigidity. Subsequent orgasm and ejaculation may be amplified due to stronger contraction of the bulbospongiosus muscle and activation of higher ejaculatory centers.

T3 Method helps to achieve satisfactory erection

Erectile function in the penis is regulated by autonomic (parasympathetic and sympathetic) and somatic (sensory and motor) pathways to the erectile tissues and perineal striated muscles..

The primary mediator of parasympathetic input is NO. The ability of NO, a highly reactive and unstable gas, to regulate a wide array of physiological functions Along with carbon monoxide, NO is a unique primary effector molecule with the characteristics of an intracellular second messenger. It is apparently synthesized on demand with little or no storage and it directly activates a soluble enzyme (guanylate cyclase) rather than a "traditional" receptor molecule. NO is produced by nitric oxide synthase (NOS) which utilizes the amino acid L-arginine and molecular oxygen as substrates to produce NO and L-citrulline. NO can readily cross plasma membranes to enter target cells where it binds the heme component of soluble guanylate cyclase. This activation of guanylate cyclase stimulates the production of cGMP with the resultant activation of the cGMP-dependent protein kinase that regulates the intracellular events that lead to trabecular smooth muscle relaxation. The levels of cGMP are also regulated by phosphodiesterases, which break down cGMP and terminate signalling. ERT therapy makes use of potent, selective and reversible inhibitors of phosphodiesterase type 5, the major enzyme responsible for cGMP hydrolysis in penile erectile tissue. Inhibition of this enzyme leads to the increase of intracellular cGMP levels and enhancement of smooth muscle relaxation in response to stimuli that activate the NO/cGMP pathway. Such activity may explain the successful utility of these agents in the treatment of male ED.

Reversal of male erectile dysfunction
T3 Method
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